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Reports of flat foot deformity in association with tibialis posterior pathology appeared in the literature in the 1960’s, in 1989 Johnson and Strom developed a classification for Tibialis Posterior dysfunction, based on the progression of deformity and its treatment.
Stability, flexibility and motor control of the hindfoot are very important components of the normally functioning foot. Movement of the hindfoot from eversion to inversion , alters the alignment of the chopart articulation and converts the flexible foot, useful for balance and shock absorption, into a closed pack, rigid configuration, which allows the foot to function as a mechanically effective lever. Normal hindfoot mechanics are reliant upon static (ligamentous), dynamic (musculo-tendinous) restraints, it’s function will also be affected by proximal (e.g valgus knee) and distal (e.g. elevated 1st ray) deformity. There are multiple interlinked static restraints of the hindfoot including the Deltoid ligament, Plantar Fascia, Spring Ligament, Interosseous Ligaments, cervical ligament, the inferior extensor retinaculum.
Dynamic stabilisers include the Tibialis Posterior, Tibialis Anterior, the Achilles, Peroneals, Intrinsic foot muscles and to a lesser degree the long to flexors. The Tibialis Posterior tendon is the primary muscle motor, acting to invert the hindfoot converting its function to allow the calf muscles to generate force through the foot more efficiently, and to prevent excessive eversion. Failure of either the ligamentous restraints, the joints or the muscle motors lead to dysfunction of the foot, often in association with pain.
The Tibialis Posterior muscle originates from the posterior border of the Tibia, the Interosseous Membrane and the fibula. the tendon forms in the lower third of the leg and passes within a thick retinaculum in a groove over the posterior and inferior margin of the medial malleolus. The tendon develops a major insertion onto the plantar medial aspect of the navicular tuberosity before passing plantarly with insertions into the cuneiforms, the 2-4th metatarsals and the cuboid.
The most common cause of adult acquired flat foot is a failure of the Tibialis Posterior tendon, this can fail in the midsubstance or at the insertion, where it develops tendinopathy with midsubstance splits, tears and stretching, which may ultimately result in a complete rupture or more often an elongated, dysfunctional tendon.
There is debate as to whether, the ultimate failure of the Tibialis Posterior tendon is the primary pathology, or whether this is the common end point of progressive failure of the static ligamentous constraints, leading to abnormal kinematics of the hindfoot which results to abnormal strain on the Tibialis Posterior tendon. Dyal found that 70% of patients with tibialis posterior insufficiency had a contralateral flat foot, suggesting a pre-existing tendency to over-pronation is a significant risk factor. Age hormonal and genetic factors are also likely to play a role on the condition, Kohls-Gatzoulis 2009 reported this to be more common in the over 40 population with a ratio 3:1 female: male. Various authors have shown median age ranges of between 45 and 60.

INDICATIONS:
Adult acquired flat foot, failed non-operative management. Pain, restriction of function with reduced walking distance and speed.
SYMPTOMS & EXAMINATION:
Adult acquired flat foot is a progressive condition, as the deformity progresses, the symptoms tend to change and as such it presents with a multitude of complaints.
Patients will usually with unilateral symptoms which in the main progress along the same lines though with a variable timeline. The causes are various and include recurrent overuse such as seen in a runner, a history of previous injury to the hindfoot ligaments, as part of an an acute inflammatory arthropathy or due to chronic degenerative change of the tendon. Initially, pain is felt fairly precisely behind the medial malleolus and on occasion radiating down to the tendons’ insertion into the Navicular. Pain is felt on weight bearing , possibly just on exercise, but will progress sometimes to pain at rest. As deformity progresses patients begin to develop lateral hindfoot pain classically occurs with pain at the tip of the Fibula and in the sinus tarsi, due to the valgus heel abutting the soft tissues at the Fibula tip and impingement at the angle of Gissane. Whilst the deformity remains flexible these symptoms may be reduced by mechanically offloading the tendon (with orthotics and intelligent shoewear choices, and stretching of the tight gastrocnemius-soleus complex). If progression to arthritic change occurs in the subtalar (and ultimately the ankle) joints, symptoms from these joints may dominate the clinical picture.
On examination in mild cases there will be little to find. The tendon is in most cases discreetly tender but if swelling (of the tendon and associated subcutaneous oedema) is particularly severe then it may be difficult to identify the tendon itself. The degree of medial longitudinal arch reduction will vary from none to a severe planovalgus collapse. Until a fixed deformity ensues the planovalgus foot may not be obvious unless the patient is weight-bearing. It should be appreciated that the deformity is multi-planar and comprises a reduction of the height of the medial longitudinal arch ,hindfoot valgus and abduction of the forefoot due to midfoot pronation.
A key test to do in assessing the plano-valgus foot and ankle is the single heel rise test , observing the patient from behind and noting whether the hindfoot moves appropriately into varus or remains in valgus. This differentiates a grade 1 tendinopathy in the Johnson and Strom classification (see below) which will likely respond to non-operative offloading or a more minor surgical intervention if this becomes required. The Tibialis posterior can be isolated by plantar flexing and everting the foot, power is then tested from this position against manual resistance in order to test MRC strength. The tendon is tested in an plantar flexed everted position in order to remove the effect of tibialis anterior as an inverter by reducing its lever arm.
It is imperative that achilles or Gastrocnemius tightness is assessed. This is demonstrated with the foot aligned in its “anatomical position” ( The Talar-neutral position: the position in which the talar head is maximally covered by the navicular) with the knee both flexed and extended. It should be noted whether it is it possible to dorsiflex the ankle easily beyond neutral in the sagittal plane or not. If not then a tight Gastrocnemius or Achilles tendon is diagnosed which may be a contributing factor in the deformity and may also require correction at the time of surgery. The foot sitting in a plano-valgus position will allow the Achilles to shorten and become tight over time, however in some cases the Achilles tightness may be part of the primary problem rather than a secondary effect.
It should be remembered that there are many causes of a unilateral planovalgus foot (including Lisfranc arthritis , Charcot midfoot collapse, late presentation of tarsal coalition, arthritis of the triple complex and previous Calcaneal fracture ) and other diagnoses must always be considered.
The grading system of Johnson & Strom is most often used in describing the extent of the deformity and also is used in decision making on which intervention may be used most appropriately. Stage 1 is defined by tenosynovitis of the tendon with minimal deformity or weakness of the tendon and the single heel raise test is largely normal. This is likely to respond to conservative measures or a tendon sparing procedure ( debridement). A stage 2 tendon produces the classic pes planus deformity due to its elongation and reduction in power. A single heel rise will not be possible but the subtalar joint is mobile. This is the type of tendon requiring the reconstruction described in this section if conservative management fails. In stage 3 arthritis has occurred and the valgus deformity is fixed .Surgical treatment now requires a triple fusion. In stage 4 the ankle is also involved and if surgical treatment is required this is likely to involve a true pantalar fusion.
INVESTIGATION.
Plain X-Ray: Weight-bearing films can be used to document the structural deformity if one exists but are not required to make the diagnosis. Assessment of the degree of talar un-coverage is useful for surgical decision making.
MRI:The investigation of choice for Tibialis posterior tendinosis which most objectively defines both the extent of the disease process and also the existence of intercurrent arthritic change.
Ultrasound:In patients with paratenon inflammation only this can be used very effectively to both diagnose and infiltrate local anaesthetic and steroid into the appropriate layer.
Weight bearing CT scan: A useful modality for assessing the alignment of the hindfoot, forefoot and any deformity in the medial column.
NON-OPERATIVE ALTERNATIVES.
The first line management of most patients is initially non-operative with activity modification and non steroidal anti-infammatory medications.
An off the shelf semi-rigid functional foot orthotic or custom made rigid orthotic device with a medial arch, medial hindfoot posting and a heel counter, together with activity modification is a starting point. Physiotherapy modalities including therapeutic ultrasound and a graded rehab program are appropriate. Occasionally a few weeks immobilisation and strict non-weight bearing in a cast will be of benefit especially for the acutely swollen and painful tendon.
A tall and appropriately designed post-operative boot is helpful for a number of weeks and beyond this the use of a more normal pair of boots that sit well above the ankle(for example hiking boots) , controlling hindfoot movement , help.
OPERATIVE ALTERNATIVES (or adjuncts).
Cobb (Split Tibialis anterior) tendon transfer.
Flexor Hallucis Longus tendon transfer
Cotton (medial column osteotomy )or similar
Lateral column lengthening.
Triple Fusion :Should be given consideration in the more elderly as an alternative to tendon transfer even in the absence of arthritic change.
CONTRAINDICATIONS.
The usual contra-indications of poor vascularity, poor soft tissue quality, smoking and immunosuppressive treatments that would need optimising pre surgery.

The procedure can be performed under a general or spinal anaesthetic.
A popliteal nerve block is performed prior to the procedure.
The patient is positioned supine on the table with a bolster under the ipsilateral buttock.
A thigh tourniquet is applied and inflated to 300 mmHg prior to skin preparation.

1-2 night stay
2 weeks in back-slab, during which elevation is encouraged for 50-55 minutes per hour.
Anticoagulation with low molecular weight heparin should be considered.
dressing changes at 1 & 2 weeks
Complete cast between weeks 2 to 6 & non-weight bearing.The cast is changed at 2 and 4 weeks as the foot is sequentially bought from its inverted post-operative position to plantigrade
Check X-ray at 6 weeks.
The patient is transferred into a long post-operative boot and gradual increase in weight-bearing. Patients will often complain of an increase in pain and swelling from 6-8 weeks, and should be reassured that this is a likely consequence of beginning to weight bear and increased activity, they should be advised to take analgesia where necessary, regularly ice and elevate the foot and in a gradual, incremental fashion increase activity.
Add an “off the shelf” supportive , semi-rigid 3/4 length orthotic with medial hindfoot posting , for the next 3 months.
Physio to work on ankle range, gastroc stretching, intrinsic foot exercises, ankle and subtalar balance, isolating the transferred FDL muscle and strengthening regime including gluteals and core from 6 weeks.
May be comfortable enough to make transition into stiff soled walking boot or structured running shoe by 12 weeks.
Remain in this for all weight bearing for a further 6 weeks as a minimum.
The Arthroereisis screw can be associated with sinus tarsi discomfort, also it can occasionally become displaced from the sinus tarsi, and consideration should be given to removing this at 6 months post operatively. The authors plan removal at 6 months post-op with the patient from the outset, if the implant remains asymptomatic, it can always be left in situ. Removal is achieved through the sinus tarsi incision and is reasonably straight-forward.
Orthotics may be required in some cases, over the longer term . A good functional recovery will not unusually take 6 months, and it is likely to take 12 months for full recovery, even after this patients report ongoing strengthening of the transferred muscle.

Complex reconstruction for the treatment of dorsolateral peritalar subluxation of the foot
J Bone Joint Surg. 81-A. 1999 :11; 1545-1560
B C Toolan , B J Sangeorzan , S T Hansen.
Retrospective study assessing radiographic and clinical outcome of 30 control calcaneal osteotomies, spring ligament repairs and tendon transfers, with 15 study patients who underwent the same baseline procedure with the addition of an arthroereisis screw.
The study group with the arthroereisis screw showed a significant improvement in the radiographic and clinical parameters when compared to the control group. The group conclude that the arthroereisis procedure is a useful adjunct to flat foot correction with little increased risk.
Complex reconstruction for the treatment of dorsolateral peritalar subluxation of the foot
J Bone Joint Surg. 81-A. 1999 :11; 1545-1560
B C Toolan , B J Sangeorzan , S T Hansen.
All patients had grade 2 Tibialis posterior tendinosis.
All patients underwent a tri-cortical bone block distraction fusion of the calcaneo-cuboid joint . All underwent an FDL transfer through the distal Tibialis posterior tendon from plantar to dorsal. All underwent a triple cut of the Achilles tendon. No spring ligament work was undertaken but 27 patients also required 1st TMT joint fusion , 4 naviculo-cuneiform fusion and 2 underwent both .
36 patients (& 41 operations) were treated of which 88% were painfree or less painful than pre-op.
There was high patient satisfaction rates with the outcome 33 of the 36 willing to undergoe the operation again if the same circumstances presented.
That said 20% non-union at Calcaneo-cuboid joint ,32% sural nerve complications and 71% required secondary surgery.
Correction of moderate and severe acquired flexible flatfoot with medialising calcaneal osteotomy and flexor diditorum longus transfer.
J Bone Joint Surg ,2006 .88A:1726-1734
A.M.Vora ,T.R.Tudor , B.G.Parks ,L.C.Schon
Cadaveric study, 7 pairs of limbs. Different levels of deformity created. Severe deformity may require additional operative procedures.
Dysfunction of the tendon of Tibialis Posterior.
J Bone Joint Surg (br) 2004;86-B:939-46
H.J.Trnka
A good Review Article
Arthrodesis techniques in the management of stage 2 and 3 acquired adult flatfoot deformity.
J Bone Joint Surg 2005.87-A; 8:1866-1876.
J.E.Johnson , J.r.Yu.
An excellent and well illustrated and referenced review article on the role of fusions (and osteotomies) in flat foot reconstructions after Tibialis posterior failure.
Calcaneal osteotomy and transfer of the tendon of flexor digitorum longus for stage 2 dysfunction of Tibialis posterior.
J Bone Joint Surg 2002.84-B;54-58.
J.T.Wacker ,M.S.Hennessy, T.S.Saxby.
51 patients with grade 2 dysfunction prospectively enrolled.
44 reviewed at a mean 51 months follow up. 43 Excellent or good outcome regarding pain & function using AOFAS score. Only 36 scored in the same categories for deformity correction. 2 Failures resulting in Calcaneo-cuboid fusion.
The objective alignment assessed clinically was only “Fair” in 25% of patients.
On technical points the transfer of FDL described was from plantar to dorsal navicular , then suturing the tendon back onto itself. The Tibialis posterior was radically excised and not tenodesed . The spring ligament was not tightened/plicated.
No orthotics were proscribed in the post-operative period.
Short- to mid-term outcomes following the use of an arthroereisis implant as an adjunct for correction of flexible, acquired flat foot deformity in adults.
Foot Ankle Spec 2018 Apr
Walley, Green, Hallam, Juliano, Aynardi.
Reference
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