
Learn the Talar fracture: Fixation of talar body and talar neck fractures via medial malleolar osteotomy surgical technique with step by step instructions on OrthOracle. Our e-learning platform contains high resolution images and a certified CME of the Talar fracture: Fixation of talar body and talar neck fractures via medial malleolar osteotomy surgical procedure.
In general terms, fractures of the talus can be broadly divided into low and high energy injury patterns. Examples of low energy fractures include avulsions, osteochondral fractures and talar process fractures. High energy injuries will either lead to the relatively uncommon situation of talar extrusion or, more frequently, fracturing of the neck or body of the talus. In these instances, the talar fracture is often associated with other injuries that may need more urgent treatment, but equally a significant proportion of these injuries will need emergent care because they are open.
The mechanism of injury in talar neck fractures is forced ankle dorsiflexion in combination with forefoot supination. Body fractures often occur in a similar vein but have the additional element of axial loading and the hindfoot being in varus or valgus on impact. A fracture of the talar body is differentiated from a talar neck fracture by the presence of a primary coronal plane fracture line on the inferior surface of the talus involving the posterior facet of the subtalar joint. In reality, our experience in Sheffield is that invariably, body fractures also involve the neck of the talus.
It is well appreciated that both talar fracture types are associated with a poor outcome but the prognosis of displaced body fractures is uniformly poor, even when compared to talar neck fractures. It is really important not to lose sight of the fact that there are two reasons why this is the case. Firstly, the injury to the bone (and its vascular supply) and soft tissues. This has already occurred to the patient and cannot be undone. Therefore, the second reason for poor outcome is down to the further iatrogenic insult from the surgical treatment. As a surgeon, you have control of this latter cause. Therefore, careful planning is required before embarking on any surgery and this planning needs you to consider which of the key vessels that give the talus its notoriously poor blood supply have been compromised by the initial injury. Remember that 60% of the surface of the talus is covered in articular cartilage. The remaining forty per cent is occupied by joint capsular reflections and ligament insertions and that there are no tendon origins or insertions. The vascular supply to the talus arises from anastomoses from the anterior tibial artery (36%), the posterior tibial artery (47%) and the peroneal artery (17%) with relative contributions of flow indicated in brackets. Inferiorly, there is a significant supply from the anastomoses within the tarsal canal and medially through branches lying within the deep deltoid ligament.
In the following case, a 60 year-old male fell 10 feet from a ladder onto concrete. He sustained a closed injury to his left talar neck and body and the soft tissues were not threatened by any dislocated fracture fragments. This was an isolated injury.

Indications
An acute displaced fracture of the neck and/or body of the talus. With fractures purely involving the neck of the talus, the subtalar joint is affected in Hawkins’ types 2, 3 and 4 as the joint dislocates in these injuries. With fractures of the body of the talus, fracture lines invariably propagate between both the ankle and subtalar joints. In both fracture patterns, the subtalar joint is involved and therefore this means that post-traumatic arthrosis is a possible outcome. Additionally, with increasing severity of injury with neck, body or combined fractures, there is potential compromise to the vascularity of the talus and pre-disposition to avascular necrosis of the talus.
Traditionally, it has been taught that talar neck fractures should be treated with a combined anteromedial and anterolateral approach. This has been to afford the optimal access to the talus to achieve anatomical reduction but, by not considering the vascular injury, may not be the best mantra for limiting the chances of AVN. Therefore, I rarely would advocate using both these incisions. My feeling is that it is worthwhile considering which is the most comminuted side of the talar neck as operating through this side will cause little further compromise to the remaining vascularity. Also given that almost a quarter of these fractures are associated with a medial malleolar fracture, it is worthwhile considering the use of the existing fracture line as a created osteotomy for a surgical approach.
Symptoms & Examination
A significant percentage of these injuries will be open and/or associated with other injuries. In an unconscious or ventilated patient the injury of the talus should be diagnosed during the “exposure” section of Advanced Trauma Life Support principles but can easily be missed in the absence of foot deformity or breaches to the soft tissue envelope. In a conscious patient, the foot will be very painful and bearing weight will not be possible. In an obviously dislocated talus, closed reduction techniques should be performed in emergency room if there is compromise to the soft tissues. The neurovascular status of the limb should also be assessed and noted. The ankle should be splinted in a temporary cast for analgesia and to rest the soft tissues. Any open fracture should be managed with lavage, reduction, splint age and a dose of antibiotic therapy. In the event of compromised skin from an irreducible injury, emergent access to theatre should be arranged to enable further closed or open reduction manoeuvres. In an experienced emergency department, it is worthwhile spending the extra 5 minutes to obtain a CT for the information that it provides in planning the reduction.
Investigation
Plain radiographic imaging is the usual initial mode of imaging. Dependent upon the severity of the injury and the presence of deformity, these plain film images can be difficult to interpret in a meaningful manner. My personal view, and that of my colleagues in Sheffield, is that a CT scan is mandatory. This helps detail the fracture pathoanatomy and aids in planning surgical approach and methods of fixation. MRI is of little benefit in assessing these acute injuries.
Initially, Hawkins devised a classification of talar neck fractures into three discrete groups of increasing severity. In type one injuries, there is simply a minimally displaced fracture across the neck of the talus. In type two injuries, the posterior facet of the subtalar joint is subluxated/dislocated. In type three injuries, the fracture is associated with both the posterior facet of the subtalar and ankle joint subluxation/dislocation. A fourth group was later added by Canale and Kelly which represents the most severe fracture pattern. As well as both subtalar and ankle joint subluxation/dislocation, there is dislocation of the head of the talus from the talo-navicular joint. This has been universally appreciated to be associated with a poor clinical outcome. The Hawkins/Canale classification system is useful as it reflects injury severity which is useful when counselling the patient about prognosis and their chance of functional recovery. A universally accepted classification system for talar body fractures does not exist and moreover, there is no classification system for combined fractures of the body and neck of talus.
Non-operative intervention
Non-operative intervention has a role to play when there is minimal displacement of fracture fragments. Equally, in very polytraumatised or poorly patients, it may be that the risks of surgery far outweigh the benefits. This is a very rare occurrence.
Operative alternatives
Apart from in the simplest fracture patterns, open reduction and internal fixation is the mainstay of managing these injuries. The aim is to reconstruct the shape of the talus with minimal disruption of the soft tissue envelope.
Contraindications
Be very aware of the red, swollen and unstable foot with little pain. This presentation should raise the suspicion of a neuropathic foot undergoing a Charcot process.
Open incisions to the traumatised foot in the presence of diabetes, vascular disease or metabolic compromise from steroid treatment are relative contra-indications for surgical intervention.

The patient is positioned supine on the operating table and may require a sandbag under the ipsilateral buttock so that the foot points vertically towards the ceiling which allows dual incision access. For pure medial access, the sandbag may be best placed under the contralateral buttock and for isolated lateral access, the patient can be placed in a more formal lateral position. Fluoroscopy should be available with an image intensifier and a trained radiographer.
Appropriate antibiotics are administered and a thigh tourniquet and exclusion drape are applied. The limb is prepared with Chlorhexidine from toes to tourniquet.

The patient is placed in a below the knee back slab for the first two weeks after surgery. At two weeks, the wounds are inspected and re-dressed and a complete, lightweight below-the-knee cast is applied for a further four weeks. Weight bearing is not permitted for the first six weeks after surgery and in my practice, rivaroxaban is prescribed for this duration to prevent thrombo-embolic events.
At six weeks, plain radiographs are used to assess whether the medial malleolar osteotomy has united. If it has, then the patient can start bearing weight in a walker boot for a further 6 weeks. If it hasn’t united then I would maintain the period of cast immobilisation for a further 4 weeks and perform further plain radiographs.
If at 6 weeks the osteotomy looks to have healed, then physiotherapy can be started to work on the ankle, subtalar and talo-navicular joint range of motion.
At 12 weeks, further plain radiographs are useful. This is to check for Hawkins’ sign. Hawkins described subchondral osteopenia on the A-P ankle radiograph (not the lateral view) as a positive sign of revascularisation. It represents bone turnover in this region. Some authors rely on this sign to govern when the patient can start to bear weight but in some cases of body and neck fractures, this sign may never appear. At some point a pragmatic approach is required which, in my view, is that the presence of Hawkins’ sign is good news but the absence is not necessarily bad news and the patient will need to start bearing weight at some point! Therefore, all of my patients will be bearing weight by 12 weeks regardless of the the radiographic appearances. One could argue that further imaging modalities could help govern the vascularity of the post-operative talus. Certainly, I have a low threshold for using CT to look for union but some authors advocate MRI to judge for talar viability. This necessitates the use of titanium implants to minimise artefact. For me the key is the union of the osteotomy as this provides stability to the ankle to allow weight bearing.

Vallier HA, Nork SE, Benirschke SK et al. Surgical treatment of talar body fractures. J Bone joint Surg 85A: 1716-1724, 2003.
This paper presents a large series of body fractures from a major trauma centre. They conclude that combined talar neck and body fractures have the highest chance of post-traumatic osteoarthritis. Collapse of the body of the talus was almost universal in open fractures who went on to develop AVN. Almost 90% of body fractures had the sequelae of AVN or arthrosis.
Vallier HA, Nork SE, Barei DP et al. Talar neck fractures: results and outcomes. J Bone Joint Surg 86A: 1616-1624, 2004.
From the same institution and in the same year, this study looked at the outcomes from talar neck fractures. It confirmed a linear relationship of AVN with increasing Hawkins grade but also that comminuted and open fractures were most likely to result in post-traumatic osteoarthritis.
Prasarn ML, Miller AN, Dyke JP et al. Arterial anatomy of the talus: A cadaver and gadolinium-enhanced MRI study. Foot Ankle Int 31(11): 987-993, 2010.
This study is helpful because rather than describe what the patterns of vascular supply are, it quantifies the relative contribution of the three main arteries: 47% from the posterior tibial artery, 36% anterior tibial artery and 17% from the peroneal artery.
Chen H, Liu W, Deng L, Song W. The prognostic value of the Hawkins sign and diagnostic value of MRI after talar neck fractures. Foot Ankle Int 35(12): 1255-1261, 2014.
This paper confirmed that Hawkins sign was more likely to be present in lower energy talar neck fractures. In the higher grade injuries the incidence of AVN was about 50:50 and that MRI at 12 weeks was of use in determining vascularity in the absence of radiographic Hawkins sign.
Vints W, Matricali G, Geusens E et al. Long-term outcome after operative management of talus fractures. Foot Ankle Int 39(12): 1432-1443, 2018.
With over 9-year mean follow-up, this study confirms the importance of accurate anatomical reduction in trying to lessen the degree of post-traumatic osteoarthritis but that the fracture pattern was also a significant influencing variable in outcome.
Reference
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